Study Finds Link Between Cadaver-Derived Human Growth Hormone Shots and Alzheimers Disease

Title: Medical Injections Contaminated with Alzheimer’s-Related Protein Linked to Disease Development

In a shocking discovery, five patients in the UK have developed Alzheimer’s disease as a result of contaminated injections they received during their childhood. The injections, intended to treat very short stature, were administered using human growth hormone extracted from cadavers’ pituitary glands.

Scientists have recently found that these injections were contaminated with amyloid-beta protein, a protein strongly associated with the formation of brain plaques in Alzheimer’s disease. The patients, who did not possess genetic mutations linked to early-onset dementia, began exhibiting symptoms of dementia between the ages of 38 and 55.

The revelation, suggesting a possible third way for Alzheimer’s to develop, has surprised and concerned medical professionals. The therapy, once considered safe, has now raised questions about the potential risks associated with contaminated medical products. While the study was conducted on a small scale, the findings necessitate further research and confirmation from other studies.

Although the risk of transmitting Alzheimer’s through contaminated medical products is low, patients are urged to be aware of the potential risk and seek testing and treatment if needed. It is important to note that children currently undergoing treatment for short stature are not at risk, as synthetic growth hormone has been utilized since 1985.

Between 1959 and 1985, approximately 27,000 children worldwide, including 7,700 in the US, were administered cadaver-derived growth hormone. Tragically, contamination in some of these hormone samples resulted in the transmission of another disease called Creutzfeldt-Jakob disease (CJD), similar to mad cow disease. Over 250 growth hormone patients globally have since been diagnosed with CJD.

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While the proteins responsible for causing Alzheimer’s may not be transmissible through blood transfusions or organ donations, further research is required to fully understand this aspect. The study has also raised intriguing questions about the origins of Alzheimer’s and suggests potential shared characteristics with CJD.

This alarming discovery should prompt a thorough evaluation of measures aimed at preventing inadvertent disease transmission through medical or surgical procedures. Heightened caution and stringent protocols can help safeguard future patients from similar risks.

In conclusion, the advent of contaminated injections resulting in Alzheimer’s disease development highlights the importance of diligent scrutiny and regulation within the medical field. The findings from this study should serve as a catalyst for further research while spurring an urgent need to review and strengthen safety measures in medical procedures moving forward.

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About the Author: Forrest Morton

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